Sanchís-Gomar, FabiánPérez-Quilis, CarmenLucía Mulas, Alejandro2016-05-302016-05-302016Sanchís-Gomar, F., Pérez-Quilis, C., & Lucía, A. (2016). Overexpressing FSTL1 for Heart Repair. Trends in molecular medicine, 22(5), 353-354.147149141471499Xhttp://hdl.handle.net/11268/5185Myocardial infarction (MI), a major cause of death worldwide, further impairs the inherently low regenerative capacity of cardiomyocytes, with barely 1% of cardiomyocytes per year undergoing cell division [1]. While this low division rate increases in response to MI, it does not compensate for the loss of cardiomyocytes; thus, the area of scarred (fibrotic) tissue expands and such unfavorable tissue remodeling further deteriorates cardiac contractile capacity, ultimately leading to heart failure (HF).engjournal articlerestricted accessInfarto de miocardioCorazón - EnfermedadesSistema cardiovascularEnfermedad cardiovascular