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dc.contributor.author | García Lunar, Inés | |
dc.contributor.author | Pereda, Daniel | |
dc.contributor.author | Ibáñez, Borja | |
dc.contributor.author | García Álvarez, Ana | |
dc.date.accessioned | 2021-02-15T11:32:10Z | |
dc.date.available | 2021-02-15T11:32:10Z | |
dc.date.issued | 2020 | |
dc.identifier.citation | García-Lunar, I., Pereda, D., Ibáñez, B., & García-Álvarez, A. (2020). Neurohormonal Modulation as a Therapeutic Target in Pulmonary Hypertension. Cells, 9(11), 2521. https://doi.org/10.3390/cells9112521 | spa |
dc.identifier.issn | 2073-4409 | |
dc.identifier.uri | http://hdl.handle.net/11268/9853 | |
dc.description.abstract | The autonomic nervous system (ANS) and renin-angiotensin-aldosterone system (RAAS) are involved in many cardiovascular disorders, including pulmonary hypertension (PH). The current review focuses on the role of the ANS and RAAS activation in PH and updated evidence of potential therapies targeting both systems in this condition, particularly in Groups 1 and 2. State of the art knowledge in preclinical and clinical use of pharmacologic drugs (beta-blockers, beta-three adrenoceptor agonists, or renin-angiotensin-aldosterone signaling drugs) and invasive procedures, such as pulmonary artery denervation, is provided. | spa |
dc.description.sponsorship | Fondo Europeo de Desarrollo Regional (FEDER) | spa |
dc.description.sponsorship | Instituto de SaludCarlos III - Fondo de Investigación Sanitaria (PI17/00995) e Intensificación (AES2019) | spa |
dc.description.sponsorship | Ministerio de Ciencia, Innovación y Universidades | spa |
dc.description.sponsorship | Severo Ochoa Center of Excellence (SEV-2015-0505) | spa |
dc.language.iso | eng | spa |
dc.rights | Attribution-NonCommercial-NoDerivatives 4.0 Internacional | * |
dc.rights.uri | http://creativecommons.org/licenses/by-nc-nd/4.0/ | * |
dc.subject.other | Hipertensión pulmonar | spa |
dc.subject.other | Sistema nervioso autónomo | spa |
dc.subject.other | Sistema renina-angiotensina | spa |
dc.title | Neurohormonal Modulation as a Therapeutic Target in Pulmonary Hypertension | spa |
dc.type | article | spa |
dc.description.impact | 6.600 JCR (2020) Q2, 53/195 Cell Biology|No data SJR 2020 | spa |
dc.description.impact | No data IDR 2020 | spa |
dc.identifier.doi | 10.3390/cells9112521 | |
dc.rights.accessRights | openAccess | spa |
dc.subject.unesco | Aparato respiratorio | spa |
dc.subject.unesco | Enfermedad cardiovascular | spa |
dc.description.filiation | UEM | spa |
dc.peerreviewed | Si | spa |