Mostrar el registro sencillo del ítem
dc.contributor.author | Sanchís-Gomar, Fabián | |
dc.contributor.author | Lippi, Giuseppe | |
dc.contributor.author | Lucía Mulas, Alejandro | |
dc.date.accessioned | 2016-05-19T08:10:45Z | |
dc.date.available | 2016-05-19T08:10:45Z | |
dc.date.issued | 2016 | |
dc.identifier.citation | Sanchis-Gomar, F., Lippi, G., & Lucia, A. (2016). ‘Mitotherapy’ for heart failure. Trends in molecular medicine, 22(4), 267-269. | spa |
dc.identifier.issn | 14714914 | |
dc.identifier.issn | 1471499X | |
dc.identifier.uri | http://hdl.handle.net/11268/5149 | |
dc.description.abstract | Abnormalities in mitochondrial dynamics along with those for the molecular mediators involved are presently being viewed with increased interest in the field of cardiovascular disease. Recent research highlights OPA1, a dynamin-like GTPase mediating mitochondrial fusion, as well as the 'mitoproteases' OMA1 and YME1L, as potential therapeutic targets against heart failure. | spa |
dc.description.sponsorship | Sin financiación | spa |
dc.language.iso | eng | spa |
dc.title | ‘Mitotherapy’ for heart failure | spa |
dc.type | article | spa |
dc.description.impact | 10.732 JCR (2016) Q1, 12/290 Biochemistry and Molecular Biology, 14/190 Cell Biology, 6/128 Medicine, Research and Experimental | spa |
dc.description.impact | 5.052 SJR (2016) Q1, 25/423 Molecular Biology, 6/183 Molecular Medicine | spa |
dc.description.impact | No data IDR 2016 | spa |
dc.identifier.doi | 10.1016/j.molmed.2016.02.007 | |
dc.rights.accessRights | closedAccess | spa |
dc.subject.uem | Corazón | spa |
dc.subject.uem | Envejecimiento | spa |
dc.subject.unesco | Enfermedad cardiovascular | spa |
dc.subject.unesco | Envejecimiento | spa |
dc.description.filiation | UEM | spa |
dc.peerreviewed | Si | spa |
Ficheros | Tamaño | Formato | Ver |
---|---|---|---|
No hay ficheros asociados a este ítem. |